Kajian klinikal bekam merawat fibromyalgia: sakit otot sendi, letih, ganggu tidur, ingatan dan mood

Fibromyalgia adalah sakit dalam keadaan jangka panjang (kronik). Ia menyebabkan:

  • sakit pada otot dan tulang (kesakitan muskuloskeletal)
  • tenderness (tak boleh sentuh badan atau kawasan sakit, dia akan jadi sakit)
  • keletihan badan
  • gangguan tidur
  • gangguan kognitif

Walaupun doktor sendiri juga kurang memahami keadaan ini. Gejala-gejalanya meniru keadaan lain, dan tidak ada ujian sebenar untuk mengesahkan diagnosis. Akibatnya, fibromyalgia sering salah didiagnosis.

Kesakitan disebabkan oleh fibromyalgia adalah kompleks dan disebabkan oleh banyak faktor. Contohnya, fibromyalgia mungkin disebabkan oleh interaksi faktor sistem saraf pusat nociceptive, neuropathic, disregulatory, kegagalan metabolik dalam keradangan otot dan neurogenik [125].

Antara ciri – ciri positif yang menunjukkan anda menghidapi fibromyalgia adalah tahap cecair cerebrospinal yang tinggi glutamat, bahan P, sitokin radang, dan ROS(reactive oxidative stress) [126-129]. bukan kak Ros yee. Xoxo

Dalam fibromyalgia, kesakitan nokiseptor disebabkan oleh tindak balas sensori saraf oleh pelepasan bahan P dan neuropeptida lain dari ujung saraf periferal yang membawa kepada hyperalgesia disebabkan oleh pemekaan kimia pada titik tender.

* hyperalgesia :increased sensitivity to pain

In fibromyalgia, nociceptor pain is based on the excitation of the nervous sensors by the release of substance P and other neuropeptides from the peripheral nerve endings leading to hyperalgesia due to chemical sensitization at tender points.

Kesakitan neuropatik disebabkan oleh mampatan saraf seperti dalam herniasi cakera vertebral (disc tulang belakang) atau gangguan pengangkutan aksonal yang menyebabkan hipertoksitabiliti, sistem pengangkutan axonal yang terganggu dan perubahan trophik menyebabkan titik tender dalam fibromyalgia [125].

Neuropathic pain is due to nerve compression as in herniation of vertebral disks or disturbances of axonal transport causing hyperexcitability, disturbed axonal transport system and trophic changes causing tender points in fibromyalgia [125].

 

Cupping Therapy for Treatment of Fibromyalgia (in Light of Taibah Theory)

Terdapat kajian yang mengatakan cup bekam dipanaskan dengan herba sebelum dilakukan bekam angin pada 30 pesakit fibromyalgia dilaporkan berkesan untuk menurunkan tahap sakit fibromyalgia dan jumlah titik tender(tender points) (Cao et al., 2011).

Ringkasan kajian akan dilampirkan di bawah, tetapi dalam English ya. Saya tidak translate ia.

Title: Medicinal cupping therapy in 30 patients with fibromyalgia: a case series observation.

Cao, H. Hu, H, Colagiuri, B & Liu, J. (2011) Center for Evidence-Based Chinese Medicine, Beijing University of Chinese Medicine, Beijing, China.

BACKGROUND: The aim of this study was to evaluate the therapeutic effect of traditional medicinal cupping for treatment of fibromyalgia.

METHODS: A prospective case series was conducted in 30 consecutive patients with fibromyalgia at an outpatient department in a hospital in Beijing.

Patients were diagnosed according to the criteria set by the American College of Rheumatology (1990).

A bamboo cup, boiled in herbal decoction for 5 min, was applied to Ashi points for 10 min once daily for 15 days. Pain, assessed via a 10-point visual analogue scale (VAS), and the number of tender points were recorded at baseline, 5, 10, 15 days, and 2 weeks following the final treatment.

RESULTS: At baseline, the average pain was 2.63 ± 0.73 on the VAS, and patients had an average of 13.5 ± 1.66 tender points. The pain scores decreased from baseline during treatment (2.22 ± 0.77 at 5 days, 1.78 ± 0.75 at 10 days, and 1.36 ± 0.76 at 15 days) as did the number of tender points (12.57 ± 2.25 at 5 days, 11.2 ± 2.50 at 10 days, 9.33 ± 2.89 at 15 days). A total of 29 patients completed follow-up 2 weeks after the final treatment, which suggested the reduction in pain and tender points was sustained (1.31 ± 0.76 for VAS; 9.07 ± 2.96 for pain points). There were no serious adverse effects as a result of cupping.

CONCLUSIONS: Medicinal cupping therapy was associated with a reduction in fibromyalgia symptoms for both pain ratings and number of tender points. The findings from this case series should be tested in a controlled clinical trial.

Berdasarkan kajian ini dan pada teori Taibah, bekam darah dijangka menjadi rawatan yang lebih sesuai dan efektif untuk fibromyalgia kerana dapat menurunkan tekanan cairan interstisial, menurunkan sitokin radang dan bahan P (CPS melalui pencairan kemudian saliran) dan mengurangkan cairan interstitial di tapak keradangan.

(CPS ialah causative pathological substance iaitu kombinasi toksin atau kotoran yang berada dalam badan menggunakan istilah perubatan di atas).

Terdapat satu kajian mengenai pengurangan simptom fibromyalgia menggunakan rawatan yang mengurangkan cecair interstitial di tapak peradangan. Terdapat kesan anti-radang yang jelas dilihat dalam kajian ini [131].

Maksudnya, bekam darah juga dapat mengurangkan cecair interstitial di tapak peradangan, maka ianya sesuai dilakukan untuk mengurangkan dan menyembuhkan fibromyalgia.  

 

Rujukan

El Sayed SM, Mahmoud HS, Nabo MMH (2013) Medical and Scientific Bases of Wet Cupping Therapy (Al-hijamah): in Light of Modern Medicine and Prophetic Medicine. Altern Integ Med 2: 122. doi:10.4172/2327-5162.1000122

125. Zimmermann M (1991) Pathophysiological mechanisms of fibromyalgia. Clin J Pain 7 Suppl 1: S8-15.

126. Larson AA, Giovengo SL, Russell IJ, Michalek JE (2000) Changes in the concentrations of amino acids in the cerebrospinal fluid that correlate with pain in patients with fibromyalgia: implications for nitric oxide pathways. Pain 87: 201-211.

127. Sarchielli P, Mancini ML, Floridi A, Coppola F, Rossi C, et al. (2007) Increased levels of neurotrophins are not specific for chronic migraine: evidence from primary fibromyalgia syndrome. J Pain 8: 737-745.

128. Frazer KA, Wade CM, Hinds DA, Patil N, Cox DR, et al. (2004) Segmental phylogenetic relationships of inbred mouse strains revealed by fine-scale analysis of sequence variation across 4.6 mb of mouse genome. Genome Res 14: 1493-1500.

129.Ozgocmen S, Ozyurt H, Sogut S, Akyol O (2006) Current concepts in the pathophysiology of fibromyalgia: the potential role of oxidative stress and nitric oxide. Rheumatol Int 26: 585-597.131. Ali M (1999) Oxidative-dysoxygenative trigger points in fibromyalgia Pathogenesis, diagnosis and resolution. J of Integrative Med 3: 38-47.


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